Researchers at the University of Cincinnati found that new monoclonal antibody drugs may slow cognitive decline by increasing levels of a critical brain protein called amyloid-beta 42 (Aβ42 ...

While AD is primarily characterized by the accumulation of amyloid plaques and tau tangles in the brain, emerging research suggests that infectious agents, including HSV-1, may contribute to the ...

but not with [beta-amyloid]. In experiments using miniature human brain models (organoids), we saw that HSV-1 infection increases the amount of tau,” he told us. Interestingly, Shemesh said ...

In 2020, Cairn and her colleagues showed that if they infected the human brain-like tissue with HSV-1 in the lab, they developed characteristics of Alzheimer’s, including clumps of beta-amyloid.