The beta amyloid peptide (Abeta) is the primary component of the senile plaques observed in the brains of Alzheimer's patients, and this peptide is toxic to neurons and other cells in many different ...

At the same time, they caused another gene linked to neuronal cell death to express more, effectively contributing to cell death. Aside from the genes in which researchers saw alterations due to ...

Figure 3: Expression of ORP150 rescues cells from hypoxia-induced cytotoxicity. Figure 4: Overexpression of ORP150 has no effect on neuronal death induced by SNP, hydrogen peroxide or ...

In essence, these PFAS chemicals partially disabled the gene. At the same time, they caused another gene linked to neuronal cell death to express more, effectively contributing to cell death. Aside ...

Conceptual diagram summarizing the central experiment comparing cell death in amyloid-β treated neuronal cells with and without pancreatic β cell culture supernatant. Pancreas cells may produce ...

A research team led by CNR Nanotec, Lecce, Italy ─ the Institute of Nanotechnology ─ in collaboration with the European ...

New research links gut health to Alzheimer’s, using advanced imaging to reveal structural changes and potential early ...

X-ray micro- and nano-tomography can provide clues on the processes that link the neurons in the gut and brain and may trigger Alzheimer’s.